LG
Publications
- Intermittent Hypoxia Augments Pulmonary Vasoconstrictor Reactivity through PKCβ/Mitochondrial Oxidant Signaling
- Longitudinal Assessment of Cytokine Expression and Plasminogen Activation in Hantavirus Cardiopulmonary Syndrome Reveals Immune Regulatory Dysfunction in End-Stage Disease
- Hydrogen Sulfide Actions in the Vasculature
- Abstract P1109: Cholesterol Regulation of Hydrogen Sulfide-Mediated Vasodilation
- Expression of Adrenomedullin MRNA in Human Fetal Lung
- Cholesterol Disrupts H 2 S‐Mediated Vasodilation in Large Arteries
- Endothelial Cell Heterogeneity Between Conduit and Resistance Mesenteric Arteries
- Cholesterol Inhibits H 2 S‐Mediated Vasodilation
- Abstract P194: Endothelin as a Persistent Biomarker of Hypoxia
- Atrial natriuretic peptide and endothelin-3 target renal sodium-glucose cotransporter
- NFATc3 is required for chronic hypoxia‐induced pulmonary hypertension: role of BKα and β subunits
- New Evidence That Calcineurin-NFAT Signaling Is Key To PAH Pathogenesis, And Is Normally Modulated By VIP
- Actin polymerization contributes to enhanced pulmonary arterial vasoconstrictor reactivity following chronic hypoxia (1089.15)
- Activity-dependent NFATc3 nuclear accumulation in pericytes from cortical parenchymal microvessels
- Endothelial control of myogenic tone in mesenteric arteries is impaired after intermittent hypoxia exposure
- Intermittent Hypoxia Alters Small Mesenteric Artery Vasoreactivity in Mice: Role of NFATc3
- Abstract P279: Regulation Of H 2 S Production By Cystathionine Gamma-lyase In Endothelial Cells.
- PICK1 is Required for ASIC1‐Mediated Activation of NFATc3 in Pulmonary Arterial Smooth Muscle Cells
- Induction of Mucosal Tolerance to Collagen Type V Attenuates Hypoxic Pulmonary Hypertension
- Asthma and pulmonary arterial hypertension: do they share a key mechanism of pathogenesis?
- Nitric Oxide Enhances NFATc‐mediated Aquaporin‐2 Expression
- Adrenomedullin Expression in the Developing Human Fetal Lung
- Role of mitochondrial KATP channels in PKCβ‐dependent constriction of small pulmonary arteries (1090.10)
- 1700 Expression of the Nuclear Factor of Activated T Cells MRNA in Human Fetal Lung
- Effect of chronic angiotensin II inhibition on the cardiovascular system of the normal rat
- Altered T Regulatory Cell Phenotype due to Chronic Hypoxia Exposure Contributes to the Pathogenesis of Pulmonary Hypertension
- Intraluminal Pressure Is a Stimulus for NFATc3 Nuclear Accumulation
- Intermittent Hypoxia Increases Endothelin‐1‐Induced Reactive Oxygen Species Generation through PKC Signaling in Small Pulmonary Arteries
- ASIC1‐dependent Ca 2+ influx is required for NFATc3 activation in chronic hypoxia‐induced pulmonary hypertension (1090.12)
- Role of Cholesterol in the Regulation of Hydrogen Sulfide Signaling within the Vascular Endothelium
- Hydrogen sulfide and miR21 are suitable biomarkers of hypoxic exposure
- Single-cell transcriptomic heterogeneity between conduit and resistance mesenteric arteries in rats