pone.0260519.s001.pdf (4.01 MB)
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posted on 2021-11-30, 18:30 authored by Rachel E. Carlisle, Salwa Farooqi, Ming Chan Zhang, Sarah Liu, Chao Lu, Andy Phan, Elise Brimble, Jeffrey G. Dickhout(PDF)
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intraperitoneal injection ),apoptotic protein chopmolecular chaperone propertiesrenal injury causedrenal function causedprotein aggregation inducedprotective mechanisms founddiv >< papoptotic cell deathacute kidney injuryinhibit er stressinduces er stressrenal protection providedhistone hyperacetylation provideshdac inhibitors usedcommonly used modelhdac inhibitor effectsprevent er stressprotective effectscell deather stressrenal damagehistone hyperacetylationvarious mechanismschemical chaperoneinduced modelhistone deacetylationhistone deacetylasework demonstratedvitro tunicamycin modelstructural damagesteroidal antisignificant increaseprimarily duenucleoside antibioticmain objectiveinflammatory drugsincluding antibioticsendoplasmic reticulumdata suggest50 mg5 mg1 g
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