ppat.1010034.s012.docx (20.81 kB)
Clinical data of the study participants whose cells were used for the experiments in Figs 1, 2, and 3A.
journal contribution
posted on 2021-11-11, 18:37 authored by Opeyemi S. Adeniji, Leticia Kuri-Cervantes, Chenfei Yu, Ziyang Xu, Michelle Ho, Glen M. Chew, Cecilia Shikuma, Costin Tomescu, Ashley F. George, Nadia R. Roan, Lishomwa C. Ndhlovu, Qin Liu, Kar Muthumani, David B. Weiner, Michael R. Betts, Han Xiao, Mohamed Abdel-Mohsen(DOCX)
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tumor immune evasionimmune evasion mechanismknown inhibitory functioninhibitory receptor nkg2asup >- supsup >+ supselectively disrupt siglecevade nk immunosurveillancediv >< pdim supassociated hiv dnainfected cells comparedinhibitory effectsinfected cellsvivo vitro viremic infectionviral loadtarget cellssuppressed infectionsialoglycan interactionssialic acidphenotypic analysesnk subpopulationnk cellsnatural killermay contributelyse hivlower expressionhiv infectionhighly cytotoxichigher expressioncytotoxicity assayscytotoxic capacitycontaining glycansconjugating sialidaseblocking siglecbased interactionactivating receptorsactivated phenotype
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