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Additional file 2: of Transcriptional profiling of HERV-K(HML-2) in amyotrophic lateral sclerosis and potential implications for expression of HML-2 proteins

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posted on 2018-08-02, 05:00 authored by Jens Mayer, Christian Harz, Laura Sanchez, Gavin Pereira, Esther Maldener, Sara Heras, Lyle Ostrow, John Ravits, Ranjan Batra, Eckart Meese, Jose GarcĂ­a-PĂŠrez, John Goodier
Figure S1. HERV-K(HML-2) locus-specific nucleotide differences in cDNA sequences derived from gag amplicon. Figure S2. HERV-K(HML-2) locus-specific nucleotide differences in cDNA sequences derived from an env amplicon. Figure S3. HERV-K(HML-2) locus-specific nucleotide differences in cDNA sequences derived from a rec amplicon. Figure S4. HERV-K(HML-2) locus-specific nucleotide differences in cDNA sequences derived from an np9 amplicon. Figure S5. Normalized levels of HERV-W transcripts identified in various ALS and control tissue samples. Figure S6. Normalized levels of HERV-W transcripts identified in various ALS and control tissue samples. Figure S7. Expression of HERV-K(HML-2) Env protein detected with the HERM-1811-5 antibody. Figure S8. Agarose gel photos of HML-2 env-specific endpoint RT-PCRs for subsequent HML-2 transcription profiling. Figure S9. RNA qualities and correlations with GAPDH Ct-values. Figure S10. No correlation of relative HERV-K(HML-2) transcript levels with age or gender of donors. (PDF 2700 kb)

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National Institute of Neurological Disorders and Stroke

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