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IFNγ+IL-10+ CD4 T cells were Foxp3- Tr1 cells.

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posted on 2024-01-25, 18:46 authored by Miguel A. B. Mercado, Qiang Li, Charles M. Quick, Yejin Kim, Rachel Palmer, Lu Huang, Lin-Xi Li

CD4 T cells from Bhlhe40-/- DLN samples in Fig 2A were analyzed for Foxp3 expression by intranuclear staining. Data are representative of two independent experiments.

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    PLOS Pathogens

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    regulatory type 1protein basic helixfemale reproductive tractdiv >< pwhereas wt cd410r increased cd4key molecular driverheightened bacterial burdens>< sup ><decreased polyfunctional cd4>- deficient cd4exhibited severe defectschlamydia muridarum chlamydia polyfunctional responseskey regulator>mice deficientusing singletcr profilingsignificant enrichmentpartially rescuedmarked increaseintracellular bacteriumhost immunitygenetic ablationfunctional blockadedifferentiation trajectorycell signaturescell responsescell polyfunctionalitycell differentiationbacterial controlautoimmune diseases

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