Table_3.xls (9.5 kB)

Interactions of high sPD-1 with high HBV viral load and copresence of these two risk factors with genotype C HBV in risks for HCC and liver cirrhosis.

dataset

posted on 2014-11-26, 02:53 authored by Hsiang-Yun Cheng, Pei-Jen Kang, Ya-Hui Chuang, Ya-Hui Wang, Meng-Chin Jan, Chih-Feng Wu, Chih-Lin Lin, Chun-Jen Liu, Yun-Fan Liaw, Shi-Ming Lin, Pei-Jer Chen, Shou-Dong Lee, Ming-Whei Yu

ORs and 95% CIs were mutually adjusted for age (continuous variable), cigarette smoking (yes or no), alcohol consumption (yes or no), first-degree family history of HCC (yes or no), and BMI (≥25 or <25 kg/m²).

84 subjects with missing data on ultrasonography measurement of liver during follow-up were excluded from analysis.

33 subjects (2 HCC cases and 31 non-HCC subjects) with missing data on HBV genotype were excluded from analysis.

113 subjects were excluded due to missing data on follow-up ultrasonography measurement of liver and/or HBV genotype (80 were missing on follow-up ultrasonography measurement alone; 29 were missing on HBV genotype alone; 4 were missing on both variables).

AP, attributable proportion due to interaction; CI, confidence interval; HCC, hepatocellular carcinoma; LC, liver cirrhosis; OR, odds ratios; RERI, relative excess risk due to interaction.

Interactions of high sPD-1 with high HBV viral load and copresence of these two risk factors with genotype C HBV in risks for HCC and liver cirrhosis.