Fig_9.tif (1.6 MB)
ATWLPPR inhibited the diabetes-induced up-regulation of inflammatory proteins in the retina.
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posted on 2015-11-10, 04:13 authored by Jun Wang, Shuaiwei Wang, Mengling Li, Dongdong Wu, Fang Liu, Ruisheng Yang, Shaoping Ji, Ailing Ji, Yanzhang Li(A) Representative immunohistochemical images for ICAM-1 (a, b, c), GFAP (d, e, f), and VEGF (g, h, i) staining on retinal sections. Arrows, immune-positive. Scale Bar = 50 mm, 200X. GCL, ganglion cell layer; IPL, inner plexiform layer; INL, inner nuclear layer; OPL, outer plexiform layer; ONL, outer nuclear layer. (B) Western blot analysis of the GFAP, VEGF and ICAM-1 expression levels in retinas. D, Diabetic. *P < 0.05, versus the Normal group; #P < 0.05, versus the D+saline group.
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VEGFR 2DB saline groupoccludin expressionATWLPPR peptideVascular IntegrityffaSTZ injectionNormal groupATWLPPR treatment groupoxidative stress levelextravasated albuminoccludin degradationtransmembrane glycoproteinoxidase stress levelinjuryOxidative Stress NeuropilinNrp 1inhibitorergC 57BL micedrgfapicamdiabetes
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