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Additional file 1: Figure S1. of Distinct deposition of amyloid-β species in brains with Alzheimer’s disease pathology visualized with MALDI imaging mass spectrometry

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posted on 2017-10-16, 05:00 authored by Nobuto Kakuda, Tomohiro Miyasaka, Noriyuki Iwasaki, Takashi Nirasawa, Satoko Wada-Kakuda, Junko Takahashi-Fujigasaki, Shigeo Murayama, Yasuo Ihara, Masaya Ikegawa
MALDI-IMS for SP free control subjects. Figure S2. IHC for Aβ40 (BA27) or Aβ42 (BC05) in brain with AD and CAA. Figure S3. MALDI-IMS of AD brain (No.3) merged on optic density figure. Figure S4. High resolution (20 μm) figure of MALDI-IMS for various C-terminal truncated Aβ in AD with severe CAA.Figure S5. MALDI-IMS for various N-terminal truncated and modified Aβs in AD with moderate CAA. Figure S6. MALDI-IMS for various N-terminal truncated and modified Aβs in AD with severe CAA. Figure S7. Anti-Aβ41 antibody characterization. Supplementary Table S1. (PDF 1494 kb)

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Scientific Research on Innovative Areas

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    Acta Neuropathologica Communications

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    Keywords

    Amyloid βAlzheimer’s diseaseCerebral amyloid angiopathyImaging mass spectrometryC- and N-terminal variations of AβSenile plaquesγ-secretasePerivascular space

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