Daria Ilatovskaya

Publications

• Recent advances in understanding ion transport mechanisms in polycystic kidney disease
• Intact mitochondrial substrate efflux is essential for prevention of tubular injury in a sex-dependent manner
• Functional role of histamine receptors in the renal cortical collecting duct cells
• Effects of elevation of ANP and its deficiency on cardiorenal function
• Epithelial Sodium Channel Alpha Subunit (αENaC) Is Associated with Inverse Salt Sensitivity of Blood Pressure.
• Editorial: Role of Molecular Modulators in Combatting Cardiac Injury and Disease: Prevention, Repair and Regeneration.
• Nitric-Oxide-Mediated Signaling in Podocyte Pathophysiology.
• Insights Into the Molecular Mechanisms of Polycystic Kidney Diseases.
• Chronic Porphyromonas gingivalis lipopolysaccharide induces adverse myocardial infarction wound healing through activation of CD8+ T cells.
• FGF21 prevents low-protein diet-induced renal inflammation in aged mice.
• Comprehensive assessment of mitochondrial respiratory function in freshly isolated nephron segments.
• Practical notes on popular statistical tests in renal physiology
• Immune regulation of cardiac fibrosis post myocardial infarction.
• Sex differences in renal mitochondrial function: a hormone-gous opportunity for research.
• NOX4-dependent regulation of ENaC in hypertension and diabetic kidney disease.
• Differential effects of low-dose sacubitril and/or valsartan on renal disease in salt-sensitive hypertension.
• Cell free DNA as a diagnostic and prognostic marker for cardiovascular diseases.
• Adaptive immunity-driven inflammation and cardiovascular disease.
• Progression of diabetic kidney disease in T2DN rats.
• Regulation of mitochondria function by natriuretic peptides.
• CD8+ T-cells negatively regulate inflammation post-myocardial infarction.
• The exocyst acting through the primary cilium is necessary for renal ciliogenesis, cystogenesis, and tubulogenesis.
• Salt-deficient diet exacerbates cystogenesis in ARPKD via epithelial sodium channel (ENaC).
• Renal Glomerular Mitochondria Function in Salt-Sensitive Hypertension.
• Characterization of purinergic receptor expression in ARPKD cystic epithelia.
• Renal sodium transport in renin-deficient Dahl salt-sensitive rats.
• Protective role of Trpc6 knockout in the progression of diabetic kidney disease.
• An Offer We Cannot Refuse: Cell-Free DNA as a Novel Biomarker of Myocardial Infarction.
• Two-photon imaging of endothelin-1-mediated intracellular Ca(2+) handling in smooth muscle cells of rat renal resistance arteries.
• A NOX4/TRPC6 Pathway in Podocyte Calcium Regulation and Renal Damage in Diabetic Kidney Disease.
• Role of the Scaffold Protein MIM in the Actin-Dependent Regulation of Epithelial Sodium Channels (ENaC).
• Nitric oxide production by glomerular podocytes.
• Essential role of Kir5.1 channels in renal salt handling and blood pressure control.
• Monitoring undergraduate student needs and activities at Experimental Biology: APS pilot survey.
• Lack of Effects of Metformin and AICAR Chronic Infusion on the Development of Hypertension in Dahl Salt-Sensitive Rats.
• The Role of Angiotensin II in Glomerular Volume Dynamics and Podocyte Calcium Handling.
• Protease-activated receptors in kidney disease progression.
• High salt diet and caffeine: food for thought.
• Functional and therapeutic importance of purinergic signaling in polycystic kidney disease.
• Chronic cathepsin inhibition by E-64 in Dahl salt-sensitive rats.
• Podocyte injury in diabetic nephropathy: implications of angiotensin II-dependent activation of TRPC channels.
• Implementing Patch Clamp and Live Fluorescence Microscopy to Monitor Functional Properties of Freshly Isolated PKD Epithelium.
• Single-channel Analysis and Calcium Imaging in the Podocytes of the Freshly Isolated Glomeruli.
• TRPC6 channel as an emerging determinant of the podocyte injury susceptibility in kidney diseases.
• Cross-talk between insulin and IGF-1 receptors in the cortical collecting duct principal cells: implication for ENaC-mediated Na+ reabsorption.
• Impaired epithelial Na+ channel activity contributes to cystogenesis and development of autosomal recessive polycystic kidney disease in PCK rats.
• Angiotensin II has acute effects on TRPC6 channels in podocytes of freshly isolated glomeruli.
• Pharmacological characterization of the P2 receptors profile in the podocytes of the freshly isolated rat glomeruli.
• Arp2/3 complex inhibitors adversely affect actin cytoskeleton remodeling in the cultured murine kidney collecting duct M-1 cells.
• The implications of histamine metabolism and signaling in renal function
• Epidermal growth factors in the kidney and relationship to hypertension.
• Deficiency of renal cortical EGF increases ENaC activity and contributes to salt-sensitive hypertension.
• Real-time electrochemical detection of ATP and H₂O₂ release in freshly isolated kidneys.
• Regulation of ENaC in mice lacking renal insulin receptors in the collecting duct.
• Single-channel analysis of TRPC channels in the podocytes of freshly isolated Glomeruli.
• G-protein signaling modulator 1 deficiency accelerates cystic disease in an orthologous mouse model of autosomal dominant polycystic kidney disease.
• ROS production as a common mechanism of ENaC regulation by EGF, insulin, and IGF-1.
• Regulation of TRPC6 Channels by Non-Steroidal Anti-Inflammatory Drugs.
• Angiotensin II increases activity of the epithelial Na+ channel (ENaC) in distal nephron additively to aldosterone.
• Effects of cytochrome P-450 metabolites of arachidonic acid on the epithelial sodium channel (ENaC).
• Cortical actin binding protein cortactin mediates ENaC activity via Arp2/3 complex.
• NSAIDs acutely inhibit TRPC channels in freshly isolated rat glomeruli.
• Novel role of Rac1/WAVE signaling mechanism in regulation of the epithelial Na+ channel.
• The actin cytoskeleton and small G protein RhoA are not involved in flow-dependent activation of ENaC.
• Endothelin-1 inhibits the epithelial Na+ channel through betaPix/14-3-3/Nedd4-2.
• Intact cytoskeleton is required for small G protein dependent activation of the epithelial Na+ channel.
• Probenecid slows disease progression in a murine model of autosomal dominant polycystic kidney disease
• Transcriptomic changes in glomeruli in response to a high salt challenge in the Dahl SS rat
• Rodent models of AKI and AKI-CKD transition: an update in 2024
• Chronic Porphyromonas gingivalis lipopolysaccharide induces adverse myocardial infarction wound healing through activation of CD8+ T cells.
• Calcium signalling and transport in the kidney
• Inhibition of neprilysin with sacubitril without RAS blockage aggravates renal disease in Dahl SS rats
• Renal histaminergic system and acute effects of histamine receptor 2 blockade on renal damage in the Dahl salt-sensitive rat
• Characterization of purinergic receptor 2 signaling in podocytes from diabetic kidneys
• The actin binding protein profilin 1 is critical for mitochondria function
• β-Arrestin pathway activation by selective ATR1 agonism promotes calcium influx in podocytes, leading to glomerular damage
• Role of the scaffold protein MIM in the actin-dependent regulation of Epithelial Sodium Channels (ENaC)
• Renal Epithelial Mitochondria: Implications for Hypertensive Kidney Disease
• Regulation of TRPC6 Channels by non-steroid anti-inflammatory drugs
• Mechanism of epithelial sodium channel (ENaC) regulation by cortactin: Involvement of dynamin
• Mechanisms of epithelial sodium channel (ENaC) regulation by cortactin: Involvement of dynamin
• Cilia-deficient renal tubule cells are primed for injury with mitochondrial defects and aberrant tryptophan metabolism
• The role of polyamine metabolism in cellular function and physiology
• Commentary: the perspectives of harnessing the power of scattered tubular-like cells for renal repair
• An update on the role of sex hormones in the function of the cardiorenal mitochondria
• Guidelines for sex-specific considerations to improve rigor in renal research, and how we got there
• Endothelial Cell-Selective Adhesion Molecule Deficiency Exhibit Increased Pulmonary Vascular Resistance Due to Impaired Endothelial Nitric Oxide Signaling
• Guidelines for sex-specific considerations to improve rigor in renal research and how we got there
• Endothelial cell-selective adhesion molecule deficiency exhibits increased pulmonary vascular resistance due to impaired endothelial nitric oxide signaling