Aida Hansen

Publications

• Spontaneous Regression of Hepatocellular Carcinoma: When the Immune System Stands Up to Cancer
• MYC Overexpression Drives Immune Evasion in Hepatocellular Carcinoma That Is Reversible through Restoration of Proinflammatory Macrophages
• Data from MYC Overexpression Drives Immune Evasion in Hepatocellular Carcinoma That Is Reversible through Restoration of Proinflammatory Macrophages
• Supplementary Tables from MYC Overexpression Drives Immune Evasion in Hepatocellular Carcinoma That Is Reversible through Restoration of Proinflammatory Macrophages
• Supplementary Figures from MYC Overexpression Drives Immune Evasion in Hepatocellular Carcinoma That Is Reversible through Restoration of Proinflammatory Macrophages
• Supplementary Figures from MYC Overexpression Drives Immune Evasion in Hepatocellular Carcinoma That Is Reversible through Restoration of Proinflammatory Macrophages
• Supplementary Tables from MYC Overexpression Drives Immune Evasion in Hepatocellular Carcinoma That Is Reversible through Restoration of Proinflammatory Macrophages
• Bulk properties of the lipid bilayer are not essential for the thermal stability of Na,K-ATPase from shark rectal gland or pig kidney
• MYC functions as a switch for natural killer cell-mediated immune surveillance of lymphoid malignancies
• T-cell derived extracellular vesicles prime macrophages for improved STING based cancer immunotherapy
• Baseline serum levels of IgA anti-cyclic citrullinated protein antibodies in early rheumatoid arthritis predict radiographic progression after 11 years of treatment: a secondary analysis of the CIMESTRA study
• Cholesterol-binding motifs in STING that control endoplasmic reticulum retention mediate anti-tumoral activity of cholesterol-lowering compounds
• CD46 is a potent co-stimulatory receptor for expansion of human IFN-γ-producing CD8 + T cells
• ANTI-GALECTIN-9 ANTIBODY AS A NOVEL TREATMENT OPTION IN RHEUMATOID ARTHRITIS TARGETING PATHOGENIC FIBROBLAST-LIKE SYNOVIOCYTES
• The MYC oncogene — the grand orchestrator of cancer growth and immune evasion
• Data from MYC Overexpression Drives Immune Evasion in Hepatocellular Carcinoma That Is Reversible through Restoration of Proinflammatory Macrophages
• T-cell immunoglobulin and mucin domain 3 is upregulated in rheumatoid arthritis, but insufficient in controlling inflammation
• The DR6 protein from human herpesvirus-6B induces p53-independent cell cycle arrest in G(2)/M
• Non-random pairing of CD46 isoforms with skewing towards BC2 and C2 in activated and memory/effector T cells
• T-cell immunoglobulin and mucin domain 3 is upregulated in rheumatoid arthritis, but insufficient in controlling inflammation
• CD46 activation induces distinct CXCL-10 response in monocytes and monocyte-derived dendritic cells
• T-cell derived extracellular vesicles prime macrophages for improved STING based cancer immunotherapy
• Programmed death ligand 2 - A link between inflammation and bone loss in rheumatoid arthritis
• The DR6 protein from human herpesvirus-6B induces p53-independent cell cycle arrest in G2/M
• Extracellular Vesicles Transfer the Receptor Programmed Death-1 in Rheumatoid Arthritis
• Lymphocyte activation gene 3 is increased and affects cytokine production in rheumatoid arthritis
• Spatial analysis reveals targetable macrophage-mediated mechanisms of immune evasion in hepatocellular carcinoma minimal residual disease
• Non-random pairing of CD46 isoforms with skewing towards BC2 and C2 in activated and memory/effector T cells
• Divergent tropism of HHV-6AGS and HHV-6BPL1 in T cells expressing different CD46 isoform patterns