Lactobacillus acidophilus attenuated TBI-mediated increase of MLCP.
The interaction of Lactobacillus acidophilus and time on MLCP protein concentrations was not significant. (A) ELISA analysis showed that the concentrations of MLCP in intestinal smooth muscle were significantly increased after TBI, **P < 0.01 compared with control. Lactobacillus acidophilus significantly attenuated TBI-mediated increase of MLCP, #P < 0.05 compared with TBI. (B) Immunohistochemistry analysis showed that marked increases in MLCP were observed in intestinal smooth muscle after TBI, and treatment with Lactobacillus acidophilus significantly attenuated TBI-mediated increases of MLCP.