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Lineage-specific ohnologs (LSOs) retained by subfunctionalization or neofunctionalization.

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posted on 22.07.2021, 17:34 by Bruno Oliveira Silva Duran, Daniel Garcia de la serrana, Bruna Tereza Thomazini Zanella, Erika Stefani Perez, Edson Assunção Mareco, Vander Bruno Santos, Robson Francisco Carvalho, Maeli Dal-Pai-Silva

Gene duplicates showing signs of subfunctionalization and neofunctionalization as the main molecular mechanisms of retention. LSOs and singletons expression of (A) transforming growth factor 3 (tgfb3), (B) insulin like growth factor binding protein 3 (igfbp3), (C) tripartite motif containing 63 (trim63), (D) insulin like growth factor 2 (igf2), (E) rptor independent companion of mtor complex 2 (rictor), (F) follistatin (fst), (G) transforming growth factor beta 1 (tgfb1), (H) growth factor receptor bound protein 2 (grb2), (I) raf-1 proto-oncogene, serine/threonine kinase (raf1), (J) activating transcription factor 4 (atf4), (K) akt serine/threonine kinase 2 (akt2), (L) ras related GTP binding c (rragc), (M) component of inhibitor of nuclear factor kappa B kinase complex (chuk) in pacu (green lines) and tilapia (red lines) skeletal muscle are represented as relative values, Mean±SE (n = 6).

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