The original uncropped and unadjusted images relating to Figs 3A, 3C, 4C, 5C, 6A, 6C, 8A-8D and 9.
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posted on 2024-10-16, 17:40 authored by Chiung Chi Peng, Eugene Chang Yu Chen, Chang-Rong Chen, Charng-Cherng Chyau, Kuan-Chou Chen, Robert Y. PengHistory
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sterol regulatory elementrenal fatty changesrelated factor 2fatty acid synthasediv >< pchronic kidney diseasebinding protein 1srebp1 ), resultingrenal pathology inducedoxidative stress accompaniedhigh fat dietnrk52e cells treatedlipid metabolism signalingplasma tg levelsdependent kinase 4tgs within cellsintracellular lipid accumulationhigh nrf2 expressiondependently activated cyclinnrk52e cellsintracellular accumulationlipid dropletslow highlipogenic signalinginduced diabeteshigh incidencedicarbonyl stresscdk4 ),mg activatedvitro via tubular injuriesstudies revealedstill unknownpromising therapymainly contributedlipotoxicity causeddiabetes mellitusdensity lipoproteincleaved caspasecellular dysfunctionadipose tissuesabnormal increase12 weeks
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