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acsA and prpB mutants are defective in mucin cross-feeding.

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posted on 2016-08-22, 04:30 authored by Jeffrey M. Flynn, David Niccum, Jordan M. Dunitz, Ryan C. Hunter

PA14 mutants lacking genes required for acetate and propionate catabolism show a significant defect in co-culture with mucin-fermenting anaerobes. Asterisks indicate significance relative to PA14.

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    PLOS Pathogens

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    carbon fluxairway diseasepropionateCF patientsCF lung microbiotabacteriaexpectorated sputumcarbon sourcefermentative metabolismaeruginosa gene expressionvivo P16 S rRNA sequencingmucinairway damageCF pathogensenrichment culturingBacterial Mucin DegradationPseudomonas aeruginosacarbon reservoirCystic Fibrosis Airway Disease Chronic lung infections

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