Figure_7.tif (247.06 kB)
TLR4 deficiency suppressed inflammatory and fibrotic responses in diabetic kidney.
figure
posted on 2014-05-19, 03:23 authored by Jin Ma, Steven J. Chadban, Cathy Y. Zhao, Xiaochen Chen, Tony Kwan, Usha Panchapakesan, Carol A. Pollock, Huiling WuThe expression of endogenous TLR ligands was upregulated in WT-DN kidneys and TLR4−/−diabetic kidneys at week 6 compared with WT non-diabetic controls. (A). Gene expression of IL-6 (A), TNF-α (B), CCL2 (C), CXCL10 (D), TGF-β (E) and fibronectin (F) were upregulated in WT mice with diabetes, but suppressed in TLR4−/− mice with diabetes, as measured by RT-PCR. Data are presented as means ± SD; ** p<0.01; *** p<0.001. The number of animals per group was described in Figure 1.
History
Usage metrics
Categories
Keywords
cell biologySignal transductioncell signalingMembrane receptor signalingImmune receptor signalingImmunological signalingMolecular cell biologyMetabolic disordersDiabetes mellitusType 1 diabetestype 2 diabetesNephrologyChronic kidney diseaseModel organismsAnimal modelsMouse modelsdeficiencysuppressedinflammatoryfibroticresponsesdiabetic
Licence
Exports
RefWorks
BibTeX
Ref. manager
Endnote
DataCite
NLM
DC