Additional file 3: Figure S3. of MH84 improves mitochondrial dysfunction in a mouse model of early Alzheimer’s disease PohlandMaximilian PellowskaMaren AsseburgHeike HaglStephanie ReutzelMartina JoppeAljoscha BerressemDirk EckertSchamim WurglicsMario Schubert‐ZsilaveczManfred EckertGunter 2018 Western blot analysis of A brain-derived neurotrophic factor (BDNF), B Growth associated protein 43 (GAP43), and C synaptophysin in brain homogenate isolated from wild-type mice (control), Thy-1 AβPPSL (control), and MH-84-treated Thy-1 AβPPSL (MH84) mice. Tubulin (A, C) and GAPDH (B) were used as loading controls. Data represent means ± SEM. N = 11 (six females, five males); one-way ANOVA with Tukey’s multiple comparison post test (***p < 0.001, **p < 0.01, against wild-type(control); +p < 0.05 against Thy-1 AβPPSL (control)). (TIFF 12802 kb)