Supplementary Material for: RAGE Does Not Contribute to Renal Injury and Damage upon Ischemia/Reperfusion-Induced Injury
Dessing M.C.
Pulskens W.P.
Teske G.J.
Butter L.M.
van der Poll T.
Yang H.
Tracey K.J.
Nawroth P.P.
Bierhaus A.
Florquin S.
10.6084/m9.figshare.5123023.v1
https://karger.figshare.com/articles/dataset/Supplementary_Material_for_RAGE_Does_Not_Contribute_to_Renal_Injury_and_Damage_upon_Ischemia_Reperfusion-Induced_Injury/5123023
The receptor for advanced glycation end products (RAGE) mediates a variety of inflammatory responses in renal diseases, but its role in renal ischemia/reperfusion (I/R) injury is unknown. We showed that during renal I/R, RAGE ligands HMGB1 and S100B are expressed. However, RAGE deficiency does not affect renal injury and function upon I/R-induced injury.
2011-11-04 00:00:00
Ischemia/reperfusion
Kidney
RAGE
HMGB1