Supplementary Material for: RAGE Does Not Contribute to Renal Injury and Damage upon Ischemia/Reperfusion-Induced Injury Dessing M.C. Pulskens W.P. Teske G.J. Butter L.M. van der Poll T. Yang H. Tracey K.J. Nawroth P.P. Bierhaus A. Florquin S. 10.6084/m9.figshare.5123023.v1 https://karger.figshare.com/articles/dataset/Supplementary_Material_for_RAGE_Does_Not_Contribute_to_Renal_Injury_and_Damage_upon_Ischemia_Reperfusion-Induced_Injury/5123023 The receptor for advanced glycation end products (RAGE) mediates a variety of inflammatory responses in renal diseases, but its role in renal ischemia/reperfusion (I/R) injury is unknown. We showed that during renal I/R, RAGE ligands HMGB1 and S100B are expressed. However, RAGE deficiency does not affect renal injury and function upon I/R-induced injury. 2011-11-04 00:00:00 Ischemia/reperfusion Kidney RAGE HMGB1