Elsarraj, Hanan Hong, Yan Valdez, Kelli Michaels, Whitney Hook, Marcus Smith, William Chien, Jeremy Herschkowitz, Jason Troester, Melissa Beck, Moriah Inciardi, Marc Gatewood, Jason May, Lisa Cusick, Therese McGinness, Marilee Ricci, Lawrence Fan, Fang Tawfik, Ossama Marks, Jeffrey Knapp, Jennifer Yeh, Hung-Wen Thomas, Patricia Carrasco, D. Fields, Timothy Godwin, Andrew Behbod, Fariba Additional file 11: of Expression profiling of in vivo ductal carcinoma in situ progression models identified B cell lymphoma-9 as a molecular driver of breast cancer invasion Analysis of the The Cancer Genome Atlas (TCGA) data showed differentially expressed genes in BCL9-high versus BCl9-low tumors. Differential gene expression was performed between TCGA breast cancer samples with a normal range of BCL9 and samples with upregulated BCL9. A list of significant genes (1,756 down regulated 980 upregulated) was obtained with a threshold of a false discovery rate ≥0.05 and log fold change 0.26. (XLSX 1492 kb) BCl 9-low tumors;upregulated BCL 9.;XLSX 1492 kb;vivo ductal carcinoma;change 0.26.;B cell lymphoma -9;TCGA breast cancer samples;980 upregulated;BCL 9;BCL 9-high;Differential gene expression;Cancer Genome Atlas;breast cancer invasion Analysis;progression models 2015-09-17
    https://springernature.figshare.com/articles/dataset/Additional_file_11_of_Expression_profiling_of_in_vivo_ductal_carcinoma_in_situ_progression_models_identified_B_cell_lymphoma-9_as_a_molecular_driver_of_breast_cancer_invasion/4445381
10.6084/m9.figshare.c.3637697_D10.v1