10.6084/m9.figshare.c.3606797_D1.v1 Zuhair Al-Hassnan Zuhair Al-Hassnan Zarghuna Shinwari Zarghuna Shinwari Salma Wakil Salma Wakil Sahar Tulbah Sahar Tulbah Shamayel Mohammed Shamayel Mohammed Zuhair Rahbeeni Zuhair Rahbeeni Mohammed Alghamdi Mohammed Alghamdi Monther Rababh Monther Rababh Dilek Colak Dilek Colak Namik Kaya Namik Kaya Majid Al-Fayyadh Majid Al-Fayyadh Jehad Alburaiki Jehad Alburaiki Additional file 1: Table S1. of A substitution mutation in cardiac ubiquitin ligase, FBXO32, is associated with an autosomal recessive form of dilated cardiomyopathy Springer Nature 2016 Cardiomyopathy FBXO32 Ubiquitin proteasome system 2016-01-14 05:00:00 Journal contribution https://springernature.figshare.com/articles/journal_contribution/Additional_file_1_Table_S1_of_A_substitution_mutation_in_cardiac_ubiquitin_ligase_FBXO32_is_associated_with_an_autosomal_recessive_form_of_dilated_cardiomyopathy/4349804 Forty-eight OMIM genes implicated in dilated and hypertrophic familial cardiomyopathy that were screened for all the homozygous variants detected by whole exome sequencing. All of the identified 62 variants have been previously reported in the dbSNP and/or 1000 Genomes databases as polymorphisms, except for two novel variants in TNN and SGCD genes, which were found to be intronic. (DOCX 17 kb)