10.1371/journal.ppat.1005846.g005
Jeffrey M. Flynn
Jeffrey
M. Flynn
David Niccum
David
Niccum
Jordan M. Dunitz
Jordan M.
Dunitz
Ryan C. Hunter
Ryan C.
Hunter
<i>acsA</i> and <i>prpB</i> mutants are defective in mucin cross-feeding.
Public Library of Science
2016
carbon flux
airway disease
propionate
CF patients
CF lung microbiota
bacteria
expectorated sputum
carbon source
fermentative metabolism
aeruginosa gene expression
vivo P
16 S rRNA sequencing
mucin
airway damage
CF pathogens
enrichment culturing
Bacterial Mucin Degradation
Pseudomonas aeruginosa
carbon reservoir
Cystic Fibrosis Airway Disease Chronic lung infections
2016-08-22 04:30:01
Figure
https://plos.figshare.com/articles/figure/_i_acsA_i_and_i_prpB_i_mutants_are_defective_in_mucin_cross-feeding_/3750147
<p>PA14 mutants lacking genes required for acetate and propionate catabolism show a significant defect in co-culture with mucin-fermenting anaerobes. Asterisks indicate significance relative to PA14.</p>