10.1371/journal.ppat.1005846.g005 Jeffrey M. Flynn Jeffrey M. Flynn David Niccum David Niccum Jordan M. Dunitz Jordan M. Dunitz Ryan C. Hunter Ryan C. Hunter <i>acsA</i> and <i>prpB</i> mutants are defective in mucin cross-feeding. Public Library of Science 2016 carbon flux airway disease propionate CF patients CF lung microbiota bacteria expectorated sputum carbon source fermentative metabolism aeruginosa gene expression vivo P 16 S rRNA sequencing mucin airway damage CF pathogens enrichment culturing Bacterial Mucin Degradation Pseudomonas aeruginosa carbon reservoir Cystic Fibrosis Airway Disease Chronic lung infections 2016-08-22 04:30:01 Figure https://plos.figshare.com/articles/figure/_i_acsA_i_and_i_prpB_i_mutants_are_defective_in_mucin_cross-feeding_/3750147 <p>PA14 mutants lacking genes required for acetate and propionate catabolism show a significant defect in co-culture with mucin-fermenting anaerobes. Asterisks indicate significance relative to PA14.</p>